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another area we've had trouble with is hepatitis c. i had a male patient i diagnosed with hepatitis c, sent to a tertiary center where they did a recombinant dna (deoxyribonucleic acid) and confirmed type c. but a different tertiary center tested twice and it was negative.
what is the pathway of the enzymes that is making this congenital adrenal hyperplasia abnormal?
what is the angiotensin converting enzyme (ace) inhibitor of choice?
in a 47-year-old woman taking isoniazid (inh) as tuberculosis prophylaxis, should you get periodic liver enzymes?
what is the reference that said fatty infiltration of the liver is the most common cause of mildly elevated liver enzymes?
when should we be using troglitazone (rezulin) for patients with diabetes? which patients? how often to check liver enzymes?
what do you do with an angiotensin-converting-enzyme-inhibitor-induced cough?
do angiotensin converting enzyme inhibitors aggravate glaucoma (for example, plendil)?
how often do you need to check creatine phosphokinase (cpk) and liver enzymes in someone on 3-hydroxy-3-methyl-glutaryl-coenzyme a (hmg coa) reductase inhibitors?
how high is it safe to let the liver enzymes, alanine aminotransferase (alt) and aspartate aminotransferase (ast) go up when someone is on isoniazid (inh)?
should i put all diabetics on angiotensin converting enzyme (ace) inhibitors to protect their kidneys, even if they don't have hypertension?
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You asked:Which enzyme is involved in the maintenance of DNA (cytosine-5-)-methylation
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